Buch, Englisch, 218 Seiten, HC runder Rücken kaschiert, Format (B × H): 160 mm x 241 mm, Gewicht: 1120 g
Buch, Englisch, 218 Seiten, HC runder Rücken kaschiert, Format (B × H): 160 mm x 241 mm, Gewicht: 1120 g
ISBN: 978-3-540-22485-3
Verlag: Springer Berlin Heidelberg
Zielgruppe
Research
Fachgebiete
Weitere Infos & Material
Basics.- 1 Introduction: Towards a Molecular Classification of Chronic Myeloproliferative Disorders.- 2 Chronic Myeloproliferative Disorders: A Clinician’s View.- 3 Chronic Myeloproliferative Disorders: A Pathologist’s View.- 4 Cytogenetics of Myeloproliferative Disorders.- Polycythemia Vera.- 5 Classification and Molecular Biology of Polycythemias (Erythrocytoses).- 6 Characterization of PRV-1, a Novel Hematopoietic Cell Surface Marker Overexpressed in Polycythemia Rubra Vera.- 7 Impaired Expression of the Thrombopoietin Receptor, Mpl, in Polycythemia Vera.- 8 Growth Factor Signaling in Polycythemia Vera Cells: Specific Hypersensitivities to Cytokines in the Chronic Myeloproliferative Disorders.- 9 Role of the Antiapoptotic Protein Bcl-xL in the Pathogenesis of Polycythemia Vera.- 10 Involvement of Chromosome 9 and 11 in Familial and Sporadic Polycythemia Vera.- Essential Thrombocythemia.- 11 Essential Thrombocythemia: Regulation of Megakaryocytopoiesis.- 12 Hereditary Thrombocythemia.- 13 Thromboembolic Complications in Essential Thrombocythemia: the Role of the Analysis of the Platelet Proteome.- 14 Clonality Studies in Essential Thrombocythemia.- 15 Molecular and Preclinical Aspects of Anagrelide Action.- Chronic Idiopathic Myelofibrosis.- 16 Prolactin-like Protein E and Megakaryocytopoiesis.- 17 Pathophysiology and Stem Cell Biology in Myelofibrosis with Myeloid Metaplasia.- 18 Transforming Growth Factor ß1 in Myelofibrosis.- 19 Idiopathic Myelofibrosis: Evidence for Pathological Crosstalk Between Polymorphonuclear Leukocytes and Megakaryocytes.- Mastocytosis.- 20 C-kit and Mastocytosis.- 21 Tryptases, a Marker of Occult Mastocytosis.- Hypereosinophilic Syndrome.- 22 Familial Hypereosinophilic Syndrome.- 23 Mononuclear Cell Preparations from Patients with Idiopathic Hypereosinophilic Syndromes Do Not Express the Wilms’ Tumor Gene.- 24 Clonality Analysis and Hypereosinophilia.- 25 Clonal Lymphocytes in Patients with Idiopathic Hypereosinophilic Syndrome.