Bradshaw / Mattingley Clinical Neuropsychology

Behavioral and Brain Science
1. Auflage 2013
ISBN: 978-0-08-091700-9
Verlag: Elsevier Science & Techn.
Format: PDF
Kopierschutz: 1 - PDF Watermark

E-Book, Englisch, 458 Seiten, Web PDF

Clinical Neuropsychology is an up-to-the minute overview of the major and many interesting minor disorders and behavioral syndromes caused by localized brain damage or abnormal brain functioning. The text combines clinical findings with studies on normal, healthy individuals to provide a comprehensive picture of the human brain's operation and function. Biological rather than cognitive in emphasis, Clinical Neuropsychology integrates findings across a broad range of disciplines. This text serves as an up-to-date reference source for clinicians, researchers, and graduate students and as a textbook for advanced undergraduate courses on clinical neuropsychology. Coverage includes the ramifications of localized brain damage/abnormal brain functioning on emotion, thought, language, and behavior, illustrative case histories, chapter overviews, and more than 700 recent references. - More than 700 recent references - Extensive illustrations - Interesting and unusual illustrative case histories from recent literature - An overview and a list of important further readings end each chapter - Comprehensive index

John L. Bradshaw works at Monash University, Clayton, Victoria, Australia.

Autoren/Hrsg.

Bradshaw, John L.

Mattingley, Jason B.

Inhaltsverzeichnis

1;Front Cover;1
2;Clinical Neuropsychology: Behavioral and Brain Science;4
3;Copyright Page;5
4;Table of Contents;6
5;Preface;14
6;Chapter 1. An Introduction to Clinical Neuropsychology;18
6.1;Neuropsychology, Neuropsychiatry, and Behavioral Neurology;19
6.2;Important Assumptions in Neuropsychology;20
6.3;Conceptual Issues in Neuropsychology;23
6.4;Cerebral Circulation and Stroke;28
6.5;Stroke Syndromes Associated with Lesions in Major Arterial Fields;30
6.6;Neuroimaging Techniques;34
6.7;Neural Networks;38
6.8;Summary and Conclusions;39
6.9;Further Reading;41
7;Chapter 2. Disorders of Speech;42
7.1;Morphological Asymmetries in the Brain;43
7.2;Other Evidence for Evolutionary Adaptations for Language;45
7.3;Aphasia: General Considerations;48
7.4;Wernicke's Aphasia;51
7.5;Broca's Aphasia;54
7.6;Other Types of Aphasia;55
7.7;Subcortical Aphasias;59
7.8;Illustrative Case History of Paroxysmal Aphasia;62
7.9;Summary and Conclusions;64
7.10;Further Reading;66
8;Chapter 3. Disorders of Reading and Writing;67
8.1;Alexia with Agraphia;69
8.2;Alexia without Agraphia, or Pure Alexia;70
8.3;Surface Dyslexia;71
8.4;Spelling Dyslexia;71
8.5;Deep Dyslexia;72
8.6;Phonological Dyslexia;74
8.7;Literal Alexia;74
8.8;The Agraphias;74
8.9;Preservation of Implicit Understanding in Language Processing;75
8.10;The Localization of Language Sites by Positron Emission Tomography;77
8.11;The Localization of Language Sites by Stimulation Mapping;80
8.12;The Localization of Language Processes in Congenitally Deaf Users of Visual Signing;81
8.13;Summary and Conclusions;82
8.14;Further Reading;84
9;Chapter 4. A Right-Hemisphere Contribution to Language and a Calculation Deficit;85
9.1;Major Left-Hemisphere Damage in Infancy;86
9.2;Subtotal Left-Hemisphere Damage in Infancy;87
9.3;Major Left-Hemisphere Damage in Adults;87
9.4;Subtotal Left-Hemisphere Damage in Adults;87
9.5;Evidence from Deep Dyslexia;88
9.6;Commissurotomy Studies;89
9.7;Right-Hemisphere Damage in Adults;91
9.8;Acalculia;93
9.9;Summary and Conclusions;97
9.10;Further Reading;98
10;Chapter 5. Disorders of Object Recognition;100
10.1;Apperceptive Agnosia;103
10.2;Dorsal Simultanagnosia;104
10.3;Ventral Simultanagnosia;110
10.4;Associative Agnosia;111
10.5;Optic Aphasia;112
10.6;Category-Specific Deficits;115
10.7;Tactile Agnosia or Astereognosis;118
10.8;Auditory Agnosia;119
10.9;Amusia;120
10.10;Prosopagnosia;123
10.11;Color-Related Disorders;130
10.12;Disorders of Route Finding: Topographical Agnosia or Topographagnosia;131
10.13;Horologagnosia: A Recently Described Impairment of the Ability to Tell the Time;132
10.14;Disorders of Distortion and Perseveration;133
10.15;Visual or Optic Allesthesia;134
10.16;Delusional Misidentification Syndromes;134
10.17;Summary and Conclusions;136
10.18;Further Reading;139
11;Chapter 6. Disorders of Spatial Cognition;141
11.1;Clinical Descriptions of Unilateral Neglect;142
11.2;Terminology and Deficits Related to Unilateral Neglect;146
11.3;Etiology of Unilateral Neglect;151
11.4;Models of Unilateral Neglect;155
11.5;Current Issues in Unilateral Neglect;173
11.6;Recovery and Rehabilitation;191
11.7;Summary and Conclusions;200
11.8;Further Reading;203
12;Chapter 7. The Acallosal Syndrome;204
12.1;The Forebrain Commissures;205
12.2;Prior Animal Studies;207
12.3;The Human Acallosal Syndrome;208
12.4;Zaidel's "Z" Lens Technique of Prolonged Visual Lateralization;210
12.5;Subcortical Transfer of Information across the Divided Forebrain?;211
12.6;Independent Processors, Wills, and Streams of Consciousness?;214
12.7;The Congenital Acallosal Syndrome;216
12.8;Case History: Alien-Hand Syndrome;217
12.9;Summary and Conclusions;220
12.10;Further Reading;221
13;Chapter 8. Disorders of Memory;223
13.1;Working Memory;226
13.2;Regions Involved in Long-Term Memory;228
13.3;Temporal Lobe Amnesia;231
13.4;Diencephalic Amnesia and the Wernicke-Korsakoff Syndrome;232
13.5;Basal Forebrain Amnesia;239
13.6;An Overview of the Structures and Functions Involved in the Amnesic Syndrome;240
13.7;Case History: H· M. and the Lasting Consequences of Bilateral Medial Temporal Lobectomy;242
13.8;Summary and Conclusions;243
13.9;Further Reading;245
14;Chapter 9. Dementia and Alzheimer's Disease;247
14.1;Causes of Dementia;249
14.2;Alzheimer's Disease: General Considerations;250
14.3;Criteria for Diagnosis and Manifestations of Alzheimer's Disease;251
14.4;Memory Dysfunction in Alzheimer's Disease;253
14.5;Language Deficits in Alzheimer's Disease;254
14.6;Other Deficits;255
14.7;The Role of ß-Amyloid in the Etiology of Alzheimer's Disease;257
14.8;The Inheritance and Genetics of Alzheimer's Disease;259
14.9;Summary and Conclusions;263
14.10;Further Reading;265
15;Chapter 10. Movement Control I;267
15.1;Prefrontal Cortex;269
15.2;Premotor Cortex and Supplementary Motor Area;274
15.3;Primary Motor Cortex (Motor-Sensory Cortex);278
15.4;Parietal Cortex;280
15.5;Apraxia: A Disorder of Voluntary Gesture and Action;281
15.6;Summary and Conclusions;291
15.7;Further Reading;293
16;Chapter 11. Movement Control II;295
16.1;Cerebellum;295
16.2;The Basal Ganglia;299
16.3;Summary and Conclusions;305
16.4;Further Reading
;305
17;Chapter 12. Subcortical Movement Disorders I;307
17.1;Parkinson's Disease, Aging, and the Dopaminergic System;307
17.2;Positive and Negative Symptoms in Parkinson's Disease;311
17.3;Deficits in the Internal Generation of Programmed Activity;313
17.4;Cognitive Impairments;315
17.5;Disease Progression;320
17.6;Management of the Disorder;320
17.7;Causes of Parkinson's Disease;323
17.8;Summary and Conclusions;325
17.9;Further Reading;327
18;Chapter 13. Subcortical Movement Disorders II;329
18.1;Prevalence, Age of Onset, and Inheritance of Huntington's Disease
;329
18.2;Symptomatology of Huntington's
Disease;331
18.3;Cognitive Deficits in Huntington's
Disease;333
18.4;The Neuropathology of Huntington's Disease;335
18.5;Summary and Conclusions;338
18.6;Further Reading;339
19;Chapter 14. Other Movement and Neuropsychiatric
Disorders;341
19.1;Extrapyramidal Movement Disorders;342
19.2;Tic Disorders: General Features;346
19.3;Tourette's Syndrome: General Symptomatology;349
19.4;Hereditary and Environmental Factors in Tourette's Syndrome;352
19.5;Anatomical and Neurochemical Aspects of Tourette's Syndrome and Obsessive—Compulsive Disorder
;353
19.6;Obsessive—Compulsive Disorder: General Symptomatology;355
19.7;Autism;357
19.8;Other Neuropsychiatric Deficits: Aggression and Violence
;363
19.9;Affective Disorders;364
19.10;Summary and Conclusions;366
19.11;Further Reading;368
20;Chapter 15. A Disorder of Thought;369
20.1;One Disease or Many?;371
20.2;Course and Progression of Schizophrenia;373
20.3;Attentional Deficits;377
20.4;Feedback and Feed Forward in Schizophrenia;378
20.5;Brain Anomalies in Schizophrenia;379
20.6;Drug Treatment of Schizophrenia: The Dopamine Hypothesis;383
20.7;Schizophrenia as an Inherited Disorder;384
20.8;Environmental Factors;385
20.9;Case History;387
20.10;Summary and Conclusions;387
20.11;Further Reading;389
21;Chapter 16. Final Thoughts;390
22;References;398
23;Author Index;448
24;Subject Index;468